← Peptide Database/Follistatin 344
RegenerativeTier 3 β€” Animal + early human data

Follistatin 344

FST-344 Β· Follistatin Β· FS-344

Naturally occurring glycoprotein that acts as a potent antagonist of myostatin (GDF-8) and activin, thereby removing the primary brakes on muscle growth. The 344 isoform has reduced heparin binding and preferential systemic distribution versus the locally-acting 315 isoform.

πŸ’‰ SC injection🧊 Lyophilised: βˆ’20 Β°C, stable up to 2 years. Reconstituted: refrigerate (2–8 Β°C), use within 14 days. Sensitive to repeated freeze-thaw cycles.

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Mechanism of Action

Binds and neutralises myostatin and activin A with high affinity, preventing them from signalling via ActRIIB receptors. This disinhibits Akt/mTOR pathway in myocytes, allowing unconstrained protein synthesis and satellite cell activity. Concurrently reduces fat deposition via inhibition of activin-driven adipogenesis.

Clinical Applications

  • βœ“Muscle mass gain in athletes and body composition optimisation
  • βœ“Sarcopenia prevention and reversal in older adults
  • βœ“Muscle wasting conditions (cachexia, chronic disease)
  • βœ“Combined with GH secretagogues for synergistic anabolic effect

Dosing Protocol

Recommended Dosing

SC injection. 100 mcg every other day for 10 days (standard research cycle), then 30–60 day break. Some protocols use 50–100 mcg/day for 5 consecutive days. Very limited human data β€” use minimum effective dose and extended breaks.

Safety & Contraindications

Possible Side Effects

  • ⚠Excessive muscle fullness or pump (can be uncomfortable in some muscle groups)
  • ⚠Tendon stress β€” rapid muscle growth may outpace tendon adaptation
  • ⚠Unknown long-term safety profile in humans
  • ⚠Potential FSH suppression (activin normally stimulates FSH; blocking activin reduces FSH)

Contraindications

  • βœ•Active malignancy β€” myostatin inhibition may support tumour growth in some cancers
  • βœ•Fertility treatment in women β€” FSH suppression may impair follicular development
  • βœ•Existing tendinopathy β€” rapid muscle growth without tendon readiness increases injury risk

Combinations & Synergies

πŸ”— CJC-1295 + IpamorelinGH axis + myostatin inhibition β€” powerful body composition combination
πŸ”— BPC-157 + TB-500essential injury prevention add-ons when using Follistatin (tendons and connective tissue must keep pace)
πŸ”— IGF-1 LR3downstream IGF-1 signalling complements Follistatin's myostatin blockade for maximum anabolic synergy